Qiaoying Huang is an Associate Professor in the Department of Pharmacology at Zhongshan School of Medicine, Sun Yat-sen University. She earned her PhD in Pharmacology and Neurobiology from the same institution, where she also completed her postdoctoral training. Her research focuses on the pathogenic mechanisms and therapeutic targets of Parkinson’s disease, as well as neuroregenerative therapy strategies for neurodegenerative diseases. With extensive experience in neuropharmacology and experimental neuroscience, she is dedicated to advancing treatments that promote neurological repair and recovery.
Education
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Qiaoying Huang received her Bachelor of Clinical Medicine from Zhongshan School of Medicine, Sun Yat-sen University, where she studied from 2004 to 2009. She went on to pursue her PhD in Pharmacology and Neurobiology at the same institution, completing her doctoral studies in 2015 under the supervision of Professor Mingtao Li.
Experience
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Qiaoying Huang is currently an Associate Professor at the Department of Pharmacology, Zhongshan School of Medicine, Sun Yat-sen University, a position she has held since 2023. Prior to this role, she served as an Associate Researcher at the same institution from 2018 to 2023. From 2015 to 2018, she worked as a Postdoctoral Fellow in the Zhongshan School of Medicine under the supervision of Professor Guoquan Gao, gaining valuable experience in neuropharmacological research.
Awards
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Qiaoying Huang has been recognized within her field for her contributions to neuropharmacology and has received various awards and acknowledgments from her institution and academic community, reflecting her commitment to excellence in research and education
Skills and Certifications
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Her expertise spans experimental pharmacology, neurobiology, molecular neuroscience, and therapeutic development. She is skilled in a wide range of laboratory techniques related to neurodegenerative disease models, mechanistic studies, and the evaluation of therapeutic interventions.
Research Focus
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Her research interests center on the pathogenic mechanisms and therapeutic targets of Parkinson’s disease and on neuroregenerative therapy strategies for neurodegenerative diseases. She is dedicated to uncovering new insights into disease progression and developing innovative treatments to enhance neurological recovery and repair.
Conclusion
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Qiaoying Huang exemplifies the qualities of an outstanding researcher through her innovative and impactful contributions to the field of neuropharmacology, particularly in understanding and targeting Parkinson’s disease. Her robust publication record, translational research focus, and steady academic advancement demonstrate her dedication, expertise, and leadership. She is a highly deserving candidate for the Best Researcher Award, and honoring her achievements would not only recognize her personal excellence but also inspire further advancements in neurodegenerative disease research.
Publications
Rapamycin exerts neuroprotective effects by inhibiting FKBP12 instead of mTORC1 in the mouse model of Parkinson’s disease
Authors: Zeyan Zhang; Ziyue Shen; Xie Shiming; Junyu Li; Zeyu Zhang; Sheng Zhang; Bo Peng; Qianchu Huang; Mingtao Li; Shanshan Ma et al.
Journal: Neuropharmacology
An immunomodulatory role of Fc receptor γ chain independent of FcγR ligation by IgG in acute neuroinflammation triggered by MPTP intoxication
Authors: Junguo Chen; Congmin Chen; Shanshan Ma; Junyu Li; Mingtao Li; Qiaoying Huang
Journal: Neurochemistry International
Cell type- and region-specific translatomes in an MPTP mouse model of Parkinson’s disease
Authors: Qiaoying Huang; Congmin Chen; Weizhao Chen; Chaoyu Cai; Hailin Xing; Junyu Li; Mingtao Li; Shanshan Ma
Journal: Neurobiology of Disease
Repressing PTBP1 fails to convert reactive astrocytes to dopaminergic neurons in a 6-hydroxydopamine mouse model of Parkinson’s disease
Authors: Qiaoying Huang
Journal: eLife
CREB inactivation by HDAC1/PP1γ contributes to dopaminergic neurodegeneration in Parkinson’s disease
Authors: Qiaoying Huang
Journal: The Journal of Neuroscience